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The Glt1 glutamate receptor mediates the establishment and perpetuation of chronic visceral pain in an animal model of stress-induced bladder hyperalgesia

Research output: Contribution to journalArticlepeer-review

Original languageEnglish
Pages (from-to)F628-F636
JournalAmerican Journal of Physiology - Renal Physiology
Volume310
Issue number7
DOIs
StatePublished - Apr 1 2016
Externally publishedYes

ASJC Scopus Subject Areas

  • Physiology

Keywords

  • Bladder pain syndrome
  • Ceftriaxone
  • Glutamate processing
  • Hyperalgesia
  • Interstitial cystitis
  • Excitatory Amino Acid Antagonists/pharmacology
  • Gastrointestinal Motility/drug effects
  • Visceral Pain/metabolism
  • Rats
  • Rats, Inbred WKY
  • Urinary Bladder/physiopathology
  • Ceftriaxone/pharmacology
  • Excitatory Amino Acid Agonists/pharmacology
  • Animals
  • Behavior, Animal/drug effects
  • Excitatory Amino Acid Transporter 2/metabolism
  • Female
  • Spinal Cord/drug effects
  • Stress, Physiological/physiology
  • Disease Models, Animal
  • Hyperalgesia/metabolism
  • Kainic Acid/analogs & derivatives

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