Secondary necrosis is a source of proteolytically modified forms of specific intracellular autoantigens: Implications for systemic autoimmunity

Xiwei Wu; Christine Molinaro; Neal Johnson; Carlos A. Casiano

doi:10.1002/1529-0131(200111)44:11<2642::AID-ART444>3.0.CO;2-8

Secondary necrosis is a source of proteolytically modified forms of specific intracellular autoantigens: Implications for systemic autoimmunity

Xiwei Wu, Christine Molinaro, Neal Johnson, Carlos A. Casiano

Research output: Contribution to journal › Article › peer-review

Original language	English
Pages (from-to)	2642-2652
Number of pages	11
Journal	Arthritis and Rheumatism
Volume	44
Issue number	11
DOIs	https://doi.org/10.1002/1529-0131(200111)44:11<2642::AID-ART444>3.0.CO;2-8
State	Published - 2001

ASJC Scopus Subject Areas

Immunology and Allergy
Rheumatology
Immunology
Pharmacology (medical)

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10.1002/1529-0131(200111)44:11<2642::AID-ART444>3.0.CO;2-8

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title = "Secondary necrosis is a source of proteolytically modified forms of specific intracellular autoantigens: Implications for systemic autoimmunity",

author = "Xiwei Wu and Christine Molinaro and Neal Johnson and Casiano, {Carlos A.}",

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AU - Casiano, Carlos A.

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Secondary necrosis is a source of proteolytically modified forms of specific intracellular autoantigens: Implications for systemic autoimmunity

ASJC Scopus Subject Areas

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