@article{bec46eadc3f64319afc35d873e18f089,
title = "Recombinant osteopontin attenuates brain injury after intracerebral hemorrhage in mice",
keywords = "Brain edema, Collagenase, Inducible nitric oxide synthase, Intracerebral hemorrhage, Osteopontin, Brain Edema/chemically induced, Cerebral Hemorrhage/chemically induced, Matrix Metalloproteinase 9/metabolism, STAT1 Transcription Factor/metabolism, Recombinant Proteins/pharmacology, Nitric Oxide Synthase Type II/antagonists & inhibitors, Osteopontin/pharmacology, Phosphoproteins/metabolism, Behavior, Animal, Brain/drug effects, Animals, Water/metabolism, Zonula Occludens-1 Protein, Collagenases/toxicity, Membrane Proteins/metabolism, Mice, Inbred SENCAR, Mice, Disease Models, Animal",
author = "Bihua Wu and Qingyi Ma and Hidenori Suzuki and Chunhua Chen and Wenwu Liu and Jiping Tang and John Zhang",
note = "Osteopontin (OPN), an extracellular matrix glycoprotein, has been reported to inhibit inducible nitric oxide synthase (iNOS). We examined if recombinant OPN (r-OPN) inhibits iNOS and prevents brain in",
year = "2011",
month = feb,
doi = "10.1007/s12028-010-9372-z",
language = "English",
volume = "14",
pages = "109--117",
journal = "Neurocritical Care",
issn = "1541-6933",
number = "1",
}