Presenilin 1 facilitates the constitutive turnover of β-catenin: Differential activity of Alzheimer's disease-linked PS1 mutants in the β- catenin-signaling pathway

David E. Kang, Salvador Soriano, Matthew P. Frosch, Tucker Collins, Satoshi Naruse, Sangram S. Sisodia, Gil Leibowitz, Fred Levine, Edward H. Koo

Research output: Contribution to journalArticlepeer-review

Original languageEnglish
Pages (from-to)4229-4237
Number of pages9
JournalJournal of Neuroscience
Volume19
Issue number11
DOIs
StatePublished - Jun 1 1999

ASJC Scopus Subject Areas

  • General Neuroscience

Keywords

  • Alzheimer's disease
  • Glycogen synthase kinase- 3β
  • Half-life
  • Immunoprecipitation
  • Presenilin
  • Turnover
  • β-catenin

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