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Overexpression of HPV16 E6∗ alters β-integrin and mitochondrial dysfunction pathways in cervical cancer cells

Research output: Contribution to journalArticlepeer-review

Original languageAmerican English
Pages (from-to)259-274
Number of pages16
JournalCancer Genomics and Proteomics
Volume13
Issue number4
StatePublished - Jul 1 2016

ASJC Scopus Subject Areas

  • Biochemistry
  • Molecular Biology
  • Genetics
  • Cancer Research

Keywords

  • Alkaline phosphatase
  • Alternative splicing
  • Cell spreading
  • DNA damage
  • E6∗
  • Electron transport chain
  • Glutathione
  • Human papillomavirus
  • Ingenuity pathway analysis
  • Kindlin-1
  • Mitochondrial membrane depolarization
  • Mitochondrial membrane potential
  • Oxidative phosphorylation
  • Oxidative stress
  • RhoA GTPase
  • β-integrin
  • Mitochondria/metabolism
  • Oxidative Stress
  • Humans
  • Integrins/metabolism
  • Membrane Potential, Mitochondrial
  • Female
  • Reactive Oxygen Species/metabolism
  • Gene Expression
  • Signal Transduction
  • Glutathione/metabolism
  • Papillomavirus Infections/complications
  • Repressor Proteins/genetics
  • Human papillomavirus 16/genetics
  • Cell Line, Tumor
  • Proteomics/methods
  • Uterine Cervical Neoplasms/genetics
  • Oncogene Proteins, Viral/genetics
  • DNA Damage

Disciplines

  • Molecular Biology
  • Biology

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