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Gestational hypoxia increases reactive oxygen species and inhibits steroid hormone-mediated upregulation of Ca2+-activated K+ channel function in uterine arteries

Research output: Contribution to journal › Article › peer-review

Original languageEnglish
Pages (from-to)415-422
Number of pages8
JournalHypertension
Volume64
Issue number2
DOIs
StatePublished - Aug 2014

ASJC Scopus Subject Areas

  • Internal Medicine

Keywords

  • anoxia
  • oxidative stress
  • uterine artery
  • Reactive Oxygen Species/metabolism
  • Up-Regulation/drug effects
  • Estradiol/pharmacology
  • Hypoxia/metabolism
  • Potassium Channels, Calcium-Activated/metabolism
  • Progesterone/pharmacology
  • Pregnancy
  • Animals
  • Uterine Artery/drug effects
  • Female
  • Sheep

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