Delayed recanalization reduced neuronal apoptosis and neurological deficits by enhancing liver-derived trefoil factor 3-mediated neuroprotection via LINGO2/EGFR/Src signaling pathway after middle cerebral artery occlusion in rats

Research output: Contribution to journalArticlepeer-review

Original languageEnglish
Article number114607
JournalExperimental Neurology
Volume371
DOIs
StatePublished - Jan 2024

ASJC Scopus Subject Areas

  • Neurology
  • Developmental Neuroscience

Keywords

  • Apoptosis
  • Delayed recanalization
  • Ischemic stroke
  • LINGO2
  • Middle cerebral artery occlusion
  • TFF3
  • Neuroprotection
  • Brain Ischemia
  • Signal Transduction
  • Humans
  • Liver
  • Rats
  • Male
  • Neuroprotective Agents/pharmacology
  • Rats, Sprague-Dawley
  • Animals
  • Ischemic Stroke
  • Infarction, Middle Cerebral Artery/metabolism
  • Trefoil Factor-3/pharmacology
  • RNA, Small Interfering/pharmacology
  • ErbB Receptors/metabolism
  • Proto-Oncogene Proteins c-bcl-2/metabolism

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